Pten in mouse vagina

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Pten in mouse vagina

It is well known that the phosphatase and tensin homolog deleted from chromosome 10 (Pten) is a tumor suppressor and is indispensable for epithelial cell homeostasis to prevent tumor formation. Mutations in Pten are frequently found both in sporadic and hereditary cancers, and mice with Pten deletion and/or loss-offunction mutations are highly susceptible to tumor induction by aberrant phosphat...

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Physiological functions of Pten in mouse tissues.

PTEN is a tumor suppressor gene mutated in many human sporadic cancers and in hereditary cancer syndromes such as Cowden disease, Bannayan-Zonana syndrome and Lhermitte-Duclos disease. The major substrate of PTEN is PIP3, a second messenger molecule produced following PI3K activation induced by variety of stimuli. PIP3 activates the serine-threonine kinase PKB/Akt which is involved in anti-apop...

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Morphological effects of diethylstilbestrol on neonatal mouse uterus and vagina.

Administration of diethylstilbestrol (DES) to neonatal mice causes a high incidence of vaginal adenosis comparable to adenosis in women who were exposed to DES in utero. In the present study, scanning electron microscopy was used to investigate morphological events that might be involved in the development of adenosis. The effects of DES on luminal epithelia of neonatal mouse uterus, vaginal ca...

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Nanoparticle Transport from Mouse Vagina to Adjacent Lymph Nodes

To test the feasibility of localized intravaginal therapy directed to neighboring lymph nodes, the transport of quantum dots across the vaginal wall was investigated. Quantum dots instilled into the mouse vagina were transported across the vaginal mucosa into draining lymph nodes, but not into distant nodes. Most of the particles were transported to the lumbar nodes; far fewer were transported ...

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The PTEN/PI3K/AKT Pathway in vivo, Cancer Mouse Models

When PI3K (phosphatidylinositol-3 kinase) is activated by receptor tyrosine kinases, it phosphorylates PIP2 to generate PIP3 and activates the signaling pathway. Phosphatase and tensin homolog deleted on chromosome 10 dephosphorylates PIP3 to PIP2, and thus, negatively regulates the pathway. AKT (v-akt murine thymoma viral oncogene homolog; protein kinase B) is activated downstream of PIP3 and ...

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ژورنال

عنوان ژورنال: Oncoscience

سال: 2015

ISSN: 2331-4737

DOI: 10.18632/oncoscience.197